Andrew Coggan wrote:
Having an adequately-high VO2max is a necessary but not a sufficient condition for elite endurance athletic performance.
(My words, although of course I trained with, and hence learned much from, Ed.)
:)
Andrew Coggan wrote:
Having an adequately-high VO2max is a necessary but not a sufficient condition for elite endurance athletic performance.
(My words, although of course I trained with, and hence learned much from, Ed.)
:)
I have read the papers on the runners from the early 70s (where the runners were named in the paper which is so freaking cool) where we know Pre's and Shorter's testing values. I have always wondered: Could Shorter's values on the Vo2max test have been wrong? Anyone know if there are other Vo2max test results for Shorter to confirm? This is not meant as being disrespectful to the fine folks who conducted the study, but if I had seen that result based on the other results I would have checked by equipment and then my calculations.
I have worked in a lab that tested high level athletes and every now and then we would get an "odd" reading. My boss is a stickler for getting stuff right so we would run diagnostics and double check things to make sure. We even had an athlete come back once to do the same test over (and got the same value---or at least close enough considering the variations that do occur).
Hmmmmm, this paper (from 2 MDs) says that cardiac output increased after training (albeit in those with CAD).
https://www.ahajournals.org/doi/pdf/10.1161/01.CIR.42.4.611Another one
https://www.physiology.org/doi/abs/10.1152/jappl.1986.61.3.926Now a meta-analysis where Michael Joyner is an author.
https://journals.plos.org/plosone/article?id=10.1371/journal.pone.0073182Absolute and relative VO2max have there places in the discussion.
According to Claude Bouchard's work the trainability of Vo2max has a hereditary component. This may be part of the explanation for why some improve on a training plan and others do not (or not as much).
If you want a deep dive into genetics and trainability here you go
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5688475/it's genetic wrote:
Are you denying that Cardiac Output and absolute VO2 max are related?
Sure there are other variables, age, hemoconcentraton and arterio-venous difference.
Mitochondrial density is another red herring that people bring up. Does that include you?
Do you think my Absolute VO2 max:
1, went up when I trained harder in my late 20s/early 30s?
2, Stayed about the same?
3, Went down slightly?
What about my late 30s - mid 40s when I trained even harder?
I hope no one is denying that cardiac output and Vo2max are related since cardiac output is one of the factors in determining VO2max. From our friend Fick
VO2max = (stroke volume x heart rate) x a-vO2 difference.
Cardiac output is stroke volume x heart rate.
Luv2Run wrote:
More O2 can be extracted in a trained person in part because there are more red cells in a trained person (on avg), because of an increase in capillaries in the trained muscles and an increase in mitochondria in those trained muscles.
One factor affecting how much O2 of each blood litre can be extracted is also the distribution of blood, and because well-trained athletes have higher absolute cardiac output values, proportionally higher amount of all the blood can be distributed to muscles that use very effectively the "offered" oxygen.
https://www.researchgate.net/profile/Aaron_Lynn/publication/279485469/figure/fig2/AS:650033875652658@1531991561717/Schematic-illustrating-the-distribution-of-cardiac-output-during-rest-and-at-increasing.png(all the blood "demands" by the body do not rise in tandem when the exercise intensity increases)
it's genetic wrote:
So what are you disagreeing with and why?
Running is a set of skills, so is cycling. But having a VO2 max of 75 ml/kg/min is irrelevant to how much skill you have for either.
Now you are muddling things up with the introduction of "skill"...
I would not consider running economy to be a "skill" or LT to be a "skill". Now staying upright on a bike in a nasty corner--THAT is a "skill".
It improves one's RELATIVE Vo2max, but not one's ABSOLUTE Vo2max. Now losing that weight might have a performance advantage because on is hauling around less mass, but it also could go the other way in a loss of power or reduction in plasma volume depending on what sort of mass was lost and how.
Aragon wrote:
Luv2Run wrote:
More O2 can be extracted in a trained person in part because there are more red cells in a trained person (on avg), because of an increase in capillaries in the trained muscles and an increase in mitochondria in those trained muscles.
One factor affecting how much O2 of each blood litre can be extracted is also the distribution of blood, and because well-trained athletes have higher absolute cardiac output values, proportionally higher amount of all the blood can be distributed to muscles that use very effectively the "offered" oxygen.
https://www.researchgate.net/profile/Aaron_Lynn/publication/279485469/figure/fig2/AS:650033875652658@1531991561717/Schematic-illustrating-the-distribution-of-cardiac-output-during-rest-and-at-increasing.png(all the blood "demands" by the body do not rise in tandem when the exercise intensity increases)
No doubt. Part of the reason more blood can go to the muscle is that there are more capillaries to deliver it. Yes, well-trained athletes have a higher Q--no one is arguing that is not the case.
Some seem to be arguing that Q is not trainable when there is ample evidence it can be (with proper stimulus for that person).
Runningart2004 wrote:
Science stuff...
Vo2max is computed using the Fick Equation: Q + A-V o2 diff. Q is Cardiac Output, which is Stroke Volume x Heart Rate. Stroke Voume is mostly determined by the size of the left ventricular wall, which improves somewhat when a sustained endurance program is started. A-V o2 Diff is the oxygen difference between arterial and venous blood, which is a direct much of o2 uptake by the working muscles, which is improved through increased in mitochondria density in the muscle fiber and increased capillarization (more capillaries in the muscle).
Alan
Probably just a typo, but Fick is Q x a-vO2 difference, not plus.
GJ1600 wrote:
I’ve always had this question about vo2 max, and my question is, if someone had a vo2 max of 75 and they had never trained for a 5k in their entire life. Then would they be able to run a fast time? Does vo2 max measure relative fitness, or just potential? Thanks in advance for the answers:)
I wonder if the OP is more confused now or what...
it's genetic wrote:
Learneth Ernest wrote:
No.
Absolutely no.
Having a high VO2max is completely dependabt on genetics and is the least trainable variable.
Concentrate on lactate threshold, running efficiency , and speed endurance. Your VO2max comes from mommy and daddy, but mostly mommy.
Correct.
My objections here are many. VO2max can be improved (even in absolute terms) with proper training. Now that ceiling is probably set genetically, but that does not mean that I should avoid training in the ranges shown to improve Vo2max (running at around VO2max for some length of time some number of times). Even if my Vo2max does not improve (and we know that people get faster even when Vo2max does not improve) there are adaptations taking place whether they be metabolic or nervous system in nature.
Vo2max is highly dependent on genetics both in direct and indirect ways (trainability also has a genetic component).
"Concentrate" on all these factors in a well designed training program that is well-rounded. I would never tell an athlete (even a "hobby jogger") to do training at just one pace or distance all the time.
Heck, elite endurance athletes train at a high level without them or their coaches knowing much about any of these things. The sub 2 project was enlightening because those runners and their coaches had little information on these traditional lab values. Alex Hutchinson writes about even running on a treadmill for a new experience.
These things help us understand better why a runner is getting faster. Hopefully by studying these things we get a better picture of what works in training. One issue is that some respond to the training protocol in that study and others do not. The term non-responder has fallen out of favor because of the recognition that the training stimulus just might not have been appropriate for those individuals. (I now think of it is "non-responder" to those conditions).
Success is a complex interplay of factors and finding what works for the individual sitting across the coffee shop table from you is still some trial and error.
Andrew Coggan wrote:
I worked alongside cardiologists in a world-class medical school for over 10 y. None of them really knew anything about exercise physiology, just as I don't really know anything about, say, congenital heart disease.
It would be good to discuss these issues with a cardiologist who is a long term competitive runner or cyclist.
Too much of Exercise Physiology is concerned with selling people dreams of great improvements, hence hudreds, probably thousands of studies showing that training protocl x improves VO2max by y%. Whereas Cardiologists have a responsibility and accountability to help save or lengthen peoples' lives.
Andrew Coggan wrote:
One of but dozens, if not hundreds, of studies directly demonstrating an increase in stroke volume, and hence maximal cardiac output, in response to endurance exercise training:
https://www.ncbi.nlm.nih.gov/m/pubmed/1385806/
VO2 max 2.7 liters per minute???? Hmmm
That is the type of study I'm talking about, were they properly tested? A standard protocol won't work for an unfit person because they will fatigue before they reach their true max. Actually this is true for everyone in certain circumstances.
No doubt Q (cardiac output) has a genetic link, but endurance training increases blood volume both in terms of red cell mass and plasma volume. If blood volume increases then Q increases.quote]
As stroke volume goes increases, heart rate decreases to maintain (maximal) stroke volume. This is an important issue which I'm trying to explain.
And the increase in red cell mass is transitory as hemolysis increases and thus erythropoeiesis increases to compensate.
Agree about the Fick equation, but the increase in capillaries and stroke volume happen anyway and they both decrease heart rate. More blood volume is pumped as we increase training volume (short term adaption) and as we age. Old people have more capillaries and a higher stroke volume. Cardiac output is maintained but VO2 max decreases. As for increases in Mitochondria, another red herring. Mitcondrial numbers stay the same but they increase in size with high volume training. The amount of oxygen delivered stays the same.
Luv2Run wrote:
it's genetic wrote:
So what are you disagreeing with and why?
Running is a set of skills, so is cycling. But having a VO2 max of 75 ml/kg/min is irrelevant to how much skill you have for either.
Now you are muddling things up with the introduction of "skill"...
I would not consider running economy to be a "skill" or LT to be a "skill". Now staying upright on a bike in a nasty corner--THAT is a "skill".
The skill is the efficiency of movement, including the oxygen economy. That is why the OPs question needs answering with regard to fitness in running relative to oxygen uptake.
Shorter bailed on his test while Pre raged against the machine. That's why those two numbers should not be brought up constantly.
Good to see Jon Orange (it's genetic) back trolling in full force. I guess the lab and book thing aren't working out?
Surprised the same folks are getting sucked into his vortex of BS,
Read my posts more carefully. You're missing a few points.
You're not qualified to be taken seriously, Jon. You are neither a cardiologist nor an ex. Physiologist.
You probably flunked your O levels.