Lauren Fleshman tests out whistleblowing on dopers

drugsoutnow wrote:

I applaud her efforts if she is following up on this discussion. Athletes using hormone therapy as a recovery aid is getting out of hand. The US all-time lists are filling up with athletes using these drugs.

http://www.flotrack.org/coverage/250603-2013-Outdoor-Track-and-Field-on-Flotrack/video/704658-Fleshman-Mackey-and-Magness-debate-the-thyroid-issue-Part-I

Why don't you post us a compendium list of US All Time performers, with links to their public statement of "hormone therapy."

Here's the thing...go read Magness's blog article on hypothyroidism. You can't take thyroid meds and get a performance boost unless you test positive for the gene that says you have a thyroid disease and need thyroid replacement.

US All Time Top Ten Marathon

1 1 2:05:38 Khalid Khannouchi Ossining, NY 30 Flora London GBR 4/14/2002

2 2:05:56 Khannouchi (2) Ossining, NY 30 LaSalle Bank Chicago IL 10/13/2002

3 2 2:06:17 Ryan Hall (CA) Big Bear Lake, CA 25 Flora London Marathon GBR 4/13/2008

4 2:07:01 Khannouchi (3) Ossining, NY 28 LaSalle Bank Chicago IL 10/22/2000

5 2:07:04 Khannouchi (4) Ossining, NY 35 Flora London Marathon GBR 4/23/2006

6 2:08:24 Hall (2) Big Bear Lake, CA 24 Flora London Marathon GBR 4/22/2007

7 2:08:41a Hall (3) Big Bear Lake, CA 27 B.A.A. Boston MA 4/19/2010

8 2:08:44 Kannouchi (5) Ossining, NY 32 LaSalle Bank Chicago IL 10/10/2004

9 3 2:08:47a Bob Kempainen Minnetonka, MN 27 Boston MA 4/18/1994

10 4 2:08:52a Alberto Salazar Eugene, OR 23 Boston MA 4/19/1982

40% of these times were achieved by using hormone therapy as a recovery aid! (Hall and Salazar)

1) It’s a stimulant and that somehow helps performance (how?)

2) “anorexic effects” (Behind the scenes, I’ve heard rumors an athlete might stockpile their prescription and then take extra doses before competition to help lose weight which would presumably help performance)

4) The thyroid hormone might be able to boost EPO production somehow.

5) It can help recovery and VO2 max.

6) People doping on HGH will have their hormones out of wack, so they need to get on thyroid medication to try and stabilize things.

drugsout wrote:

http://www.letsrun.com/news/2013/04/hypothyroidism-the-wall-street-journal-jos-hermens-mo-farah-galen-rupp-and-the-olympics/

1) It’s a stimulant and that somehow helps performance (how?)

2) “anorexic effects” (Behind the scenes, I’ve heard rumors an athlete might stockpile their prescription and then take extra doses before competition to help lose weight which would presumably help performance)

4) The thyroid hormone might be able to boost EPO production somehow.

5) It can help recovery and VO2 max.

6) People doping on HGH will have their hormones out of wack, so they need to get on thyroid medication to try and stabilize things.

It MIGHT be able to boost EPO production SOMEHOW? Well, folks, this is first-rate science right here.

The sky is falling! The sky is falling!!

rfdsfsdflkj wrote:

It MIGHT be able to boost EPO production SOMEHOW? Well, folks, this is first-rate science right here.

Actually it is what they are doing.

You idiots hear "thyroid" and you think that's all that's going on in Rupp's endorinologist's office in HOUSTON TEXAS. I can guarantee you that thyroid medications are not the only thing going on in Rupp's bogus TU

Es.

The thyroid hormones are inextricably linked to the pituitary and hypothalamus, all of which produce hormones necessary to growth, strength and metabolism. As someone else said, once you push one button the other buttons need pushing too.

From the pituitary:

Thyroid Stimulating Hormone (TSH)

Follicle-Stimulating Hormone (FSH)

Luteinizing Hormone (LH) responsible for secreting testosterone (get where they are going yet?)

Growth Hormone (HGH)

ACTH — the adrenocorticotropic hormone (also involved in testosterone production)

From the hypothalamus:

Gonadotropin-releasing hormone (GnRH) (also involved in testosterone production)

Corticotropin-releasing hormone (CRH) acts to release ACTH

Thyroid hormone induces erythropoietin gene expression through augmented accumulation of hypoxia-inducible factor

The meds these athletes are taking ARE ON THE BANNED LIST! They are on the banned list for a reason. They are performance enhancing drugs! However, these "athletes" get a note from a doctor to circumvent the system, giving them the right to take banned performance enhancing drugs. It's no different from a Hollywood actor with a handful of prescriptions for uppers and downers for recreational/addiction purposes.

I hope Fleshman tells the WADA what is really going on with these Therapeutic Use Exemptions. The WADA's moto is "Play True." Users of hormone therapy are not playing true. Report this to the WADA yourself, and contribute to an even playing field.

These cheats are abusing the system to beat you, take your spot on teams, and steal your earnings! Fight back by demanding the WADA clean up our sport of these cheats! Voice your opinion directly to the WADA here:

http://www.wada-ama.org/en/Footer-Links/Contact/

They are the evildoers!

Everybody is doping at that level, they are just getting away with it when they can.

Back in the early 1990's a lot of runner's were using inhalers for "excercised-induced asthma".

If you were having a bad spot you went to a doctor and they would prescribe that inhalor right away. I don't think it ever was performance enhancing unless you were really asthmatic. I took it for a while just because my coach sent me to a doctor and did a treadmill test and diagnosed me as astmatic. Or just thought there was a chance I was and let's try this.

At that time it was the big thing. Why? You would have never known you had a problem unless you were running competitively at the elite level and hit a rough patch and went to a doctor.

Now we have hormone therapy for runners at that level that supposedly are deficient. Seems to me like a slippery slope.

I was a premature born 4 pound baby born in 1967 and HGH was experimental and the doctor suggested that to my mom and she declined.

Where do you draw the line in this sport?

Galen Rupp never really needed thyroid medication. His health would never have been in harm without that. He is on some drugs strictly for athletic performance reasons and that only.

This needs to be looked at.

drugsout wrote:

US All Time Top Ten Marathon

1 1 2:05:38 Khalid Khannouchi Ossining, NY 30 Flora London GBR 4/14/2002

2 2:05:56 Khannouchi (2) Ossining, NY 30 LaSalle Bank Chicago IL 10/13/2002

3 2 2:06:17 Ryan Hall (CA) Big Bear Lake, CA 25 Flora London Marathon GBR 4/13/2008

4 2:07:01 Khannouchi (3) Ossining, NY 28 LaSalle Bank Chicago IL 10/22/2000

5 2:07:04 Khannouchi (4) Ossining, NY 35 Flora London Marathon GBR 4/23/2006

6 2:08:24 Hall (2) Big Bear Lake, CA 24 Flora London Marathon GBR 4/22/2007

7 2:08:41a Hall (3) Big Bear Lake, CA 27 B.A.A. Boston MA 4/19/2010

8 2:08:44 Kannouchi (5) Ossining, NY 32 LaSalle Bank Chicago IL 10/10/2004

9 3 2:08:47a Bob Kempainen Minnetonka, MN 27 Boston MA 4/18/1994

10 4 2:08:52a Alberto Salazar Eugene, OR 23 Boston MA 4/19/1982

40% of these times were achieved by using hormone therapy as a recovery aid! (Hall and Salazar)

Really... Salazar was was using hormone therapy in 1982? For what, menopause? Seriously, do you have any proof of anything or are you just pulling stuff out of your behind?

Are you a doctor? No.

Do you have access to Rupp's thyroid gene test?

No.

Stop being stupid.

The IAAF and WADA have been all over this for longer than a decade, and there is no "there" there.

You just read sht that baits you and take the bait.

gjfgfffgj wrote:

rekrunner wrote:

Yeah. That accurately describes life -- in kindergarten.

In real life, whistle blowing may involve inside information on those that control some important aspects of your life, like income, or job, or any future career.

Anonymity is an important asset against combatting many kinds of corruption, while providing an important incentive for those to come forward with otherwise unobtainable information, avoiding obvious risks of repercussion.

I'm sorry -- are you still in kindergarten -- do I have to dumb down the mesage?

No, I'm not in kindergarten. This isn't the CIA or a murder trial. It's running. No need to be anonymous.

Certainly there is a need/value to being anonymous.

It worked in BALCO.

And, the thread or reprisals due to a leak, is a reason for anonymous tip lines.

Law enforcement allows anonymous tip lines, as does the IRS and ICE (Immigration).

Doping in running is THEFT, and anonymous tip lines should be allowed and are valuable.

Now, does that make it harder sometimes to weed through the cranks/LRC type reports, yes. Anonymous tips with enough data to make them sound credible are the ones that would be followed up on.

The best deterrent, as Dick Pound alludes to, is Harsher Penalties. Lifetime bans, and dock the nation of the drug cheat one squad member per event they test positive in, for ten years. THAT would get the attention of every freaking enabler out there.

Rupp is on a handful of drugs on the banned list. These drugs are performance enhancing drugs. That is why they are on the banned list. None of this under dispute. Rupp and Salazar have admitted this several times, and it's been widely reported in the media.

Rupp is taking PERFORMANCE ENHANCING DRUGS that are on the BANNED LIST.

THIS IS THE PROBLEM!

So it's bad to be anonymous -- according to both Mr. gjgfgfffjg and Mr. gfxgxrgx.Recent history shows Lance Armstrong retaliated at everyone who dared to not lie for him. I guess avoiding a million dollar lawsuit for telling the truth counts as a need.

gfxgxrgx wrote:It's not the tattling. It's the being anonymous that is bad.

gjfgfffgj wrote:No, I'm not in kindergarten. This isn't the CIA or a murder trial. It's running. No need to be anonymous.

Which athletes and which meds exactly?I understood thyroid medication is neither on the banned list, nor requires a TUE.Does anyone have any facts?

Hormone and Metabolic Modulators

The following are prohibited:

1. Aromatase inhibitors including, but not limited to:

aminoglutethimide

anastrozole

androsta-1,4,6-triene-3,17-dione (androstatrienedione)

4-androstene-3,6,17 trione (6-oxo)

exemestane

formestane

letrozole

testolactone.

2. Selective estrogen receptor modulators (SERMs) including, but not limited to:

raloxifene

tamoxifen

toremifene.

3. Other anti-estrogenic substances including, but not limited to:

clomiphene

cyclofenil

fulvestrant.

4. Agents modifying myostatin function(s) including, but not limited, to:

myostatin inhibitors.

5. Metabolic modulators:

Insulins

Peroxisome Proliferator Activated Receptor δ (PPARδ) agonists (e.g. GW 1516), PPARδ-AMP-activated protein kinase (AMPK) axis agonists (e.g. AICAR)

Peptide Hormones, Growth Factors and Related Substances

The following substances and their releasing factors are prohibited:

Erythropoiesis-Stimulating Agents [e.g. erythropoietin (EPO), darbepoetin (dEPO), hypoxia-inducible factor (HIF) stabilizers, methoxy polyethylene glycol-epoetin beta (CERA), peginesatide (Hematide)]

Chorionic Gonadotrophin (CG) and Luteinizing Hormone (LH) in males

Corticotrophins

Growth Hormone (GH), Insulin-like Growth Factor-1 (IGF-1), Fibroblast Growth Factors (FGFs), Hepatocyte Growth Factor (HGF), Mechano Growth Factors (MGFs), Platelet-Derived Growth Factor (PDGF), Vascular-Endothelial Growth Factor (VEGF) as well as any other growth factor affecting muscle, tendon or ligament protein synthesis/degradation, vascularisation, energy utilization, regenerative capacity or fibre type switching

and other substances with similar chemical structure or similar biological effect(s).

Anabolic agents are prohibited.

1. Anabolic Androgenic Steroids (AAS)

a. Exogenous* AAS, including:

1-androstenediol (5α-androst-1-ene-3β,17β-diol)

1-androstenedione (5α-androst-1-ene-3,17-dione)

bolandiol (estr-4-ene-3β,17β-diol)

bolasterone

boldenone

boldione (androsta-1,4-diene-3,17-dione)

calusterone

clostebol

danazol ([1,2]oxazolo[4',5':2,3]pregna-4-en-20-yn-17α-ol)

dehydrochlormethyltestosterone (4-chloro-17β-hydroxy-17α-methylandrosta-1,4-dien-3-one)

desoxymethyltestosterone (17α-methyl-5α-androst-2-en-17β-ol)

drostanolone

ethylestrenol (19-norpregna-4-en-17α-ol)

fluoxymesterone

formebolone

furazabol (17α-methyl[1,2,5]oxadiazolo[3',4':2,3]-5α-androstan-17β-ol)

gestrinone

4-hydroxytestosterone (4,17β-dihydroxyandrost-4-en-3-one)

mestanolone

mesterolone

metenolone

methandienone (17β-hydroxy-17α-methylandrosta-1,4-dien-3-one)

methandriol

methasterone (17β-hydroxy-2α,17α-dimethyl-5α-androstan-3-one)

methyldienolone (17β-hydroxy-17α-methylestra-4,9-dien-3-one)

methyl-1-testosterone (17β-hydroxy-17α-methyl-5α-androst-1-en-3-one)

methylnortestosterone (17β-hydroxy-17α-methylestr-4-en-3-one)

methyltestosterone

metribolone (methyltrienolone, 17β-hydroxy-17α-methylestra-4,9,11-trien-3-one)

mibolerone

nandrolone

19-norandrostenedione (estr-4-ene-3,17-dione)

norboletone

norclostebol

norethandrolone

oxabolone

oxandrolone

oxymesterone

oxymetholone

prostanozol (17β-[(tetrahydropyran-2-yl)oxy]-1'H-pyrazolo[3,4:2,3]-5α-androstane)

quinbolone

stanozolol

stenbolone

1-testosterone (17β-hydroxy-5α-androst-1-en-3-one)

tetrahydrogestrinone (17-hydroxy-18a-homo-19-nor-17α-pregna-4,9,11-trien-3-one)

trenbolone (17β-hydroxyestr-4,9,11-trien-3-one)

and other substances with a similar chemical structure or similar biological effect(s).

b. Endogenous** AAS when administered exogenously:

androstenediol (androst-5-ene-3β,17β-diol)

androstenedione (androst-4-ene-3,17-dione)

dihydrotestosterone (17β-hydroxy-5α-androstan-3-one)

prasterone (dehydroepiandrosterone, DHEA, 3β-hydroxyandrost-5-en-17-one)

testosterone

and their metabolites and isomers, including but not limited to:

5α-androstane-3α,17α-diol

5α-androstane-3α,17β-diol

5α-androstane-3β,17α-diol

5α-androstane-3β,17β-diol

androst-4-ene-3α,17α-diol

androst-4-ene-3α,17β-diol

androst-4-ene-3β,17α-diol

androst-5-ene-3α,17α-diol

androst-5-ene-3α,17β-diol

androst-5-ene-3β,17α-diol

4-androstenediol (androst-4-ene-3β,17β-diol)

5-androstenedione (androst-5-ene-3,17-dione)

epi-dihydrotestosterone

epitestosterone

etiocholanolone

3α-hydroxy-5α-androstan-17-one

3β-hydroxy-5α-androstan-17-one

7α-hydroxy-DHEA

7β-hydroxy-DHEA

7-keto-DHEA

19-norandrosterone

19-noretiocholanolone.

2. Other Anabolic Agents, including but not limited to:

clenbuterol

selective androgen receptor modulators (SARMs)

tibolone

zeranol

zilpaterol.

Notes

For purposes of this section:

* "exogenous" refers to a substance which is not ordinarily capable of being produced by the body naturally.

** "endogenous" refers to a substance which is capable of being produced by the body naturally.

Hide

Expand

S2. Peptide Hormones, Growth Factors and Related Substances

The following substances and their releasing factors are prohibited:

Erythropoiesis-Stimulating Agents [e.g. erythropoietin (EPO), darbepoetin (dEPO), hypoxia-inducible factor (HIF) stabilizers, methoxy polyethylene glycol-epoetin beta (CERA), peginesatide (Hematide)]

Chorionic Gonadotrophin (CG) and Luteinizing Hormone (LH) in males

Corticotrophins

Growth Hormone (GH), Insulin-like Growth Factor-1 (IGF-1), Fibroblast Growth Factors (FGFs), Hepatocyte Growth Factor (HGF), Mechano Growth Factors (MGFs), Platelet-Derived Growth Factor (PDGF), Vascular-Endothelial Growth Factor (VEGF) as well as any other growth factor affecting muscle, tendon or ligament protein synthesis/degradation, vascularisation, energy utilization, regenerative capacity or fibre type switching

and other substances with similar chemical structure or similar biological effect(s).Hide

Expand

S3. Beta-2 Agonists

Expand

S4. Hormone and Metabolic Modulators

The following are prohibited:

1. Aromatase inhibitors including, but not limited to:

aminoglutethimide

anastrozole

androsta-1,4,6-triene-3,17-dione (androstatrienedione)

4-androstene-3,6,17 trione (6-oxo)

exemestane

formestane

letrozole

testolactone.

2. Selective estrogen receptor modulators (SERMs) including, but not limited to:

raloxifene

tamoxifen

toremifene.

3. Other anti-estrogenic substances including, but not limited to:

clomiphene

cyclofenil

fulvestrant.

4. Agents modifying myostatin function(s) including, but not limited, to:

myostatin inhibitors.

5. Metabolic modulators:

Insulins

Peroxisome Proliferator Activated Receptor δ (PPARδ) agonists (e.g. GW 1516), PPARδ-AMP-activated protein kinase (AMPK) axis agonists (e.g. AICAR)

rekrunner wrote:

So it's bad to be anonymous -- according to both Mr. gjgfgfffjg and Mr. gfxgxrgx.

Recent history shows Lance Armstrong retaliated at everyone who dared to not lie for him. I guess avoiding a million dollar lawsuit for telling the truth counts as a need.

gfxgxrgx wrote:It's not the tattling. It's the being anonymous that is bad.

gjfgfffgj wrote:No, I'm not in kindergarten. This isn't the CIA or a murder trial. It's running. No need to be anonymous.

If you they had actual evidence then Lance could not have retaliated.

A lot of the elites are as clueless as us. They think they know who's doping based on speculation and results.