so many people on here as ignorant and impossible to explain things to as vent^3
i am going to say this to you, you are a f u c k ing retard, caster is not a man identifying as a woman, caster is a human being that was BORN with a GENETIC MUTATION, we are all FEMALES at first in the early stages of development in the womb yeah i know i cant call that a baby cause you liberal idiots want to call it a fetus so its ok to abort but thats another matter...
caster was born with a VAGINA, but her INTERNAL female reproductive parts where not developed but instead TESTES, if you clicked on the links i provided you would have educated yourself, but as 100% liberals do they HATE THE TRUTH and spew insults and OPINIONS and not facts
then there are some here b*tching that caster needs to take hormone therapy then compete....so taking PEDs to improve it bad but taking things that would CHANGE your natural HORMONE levels drastically is ok? you think thats ok to do? alter your bodys natural levels? you dont think there will be side effects? my guess would be depression, and suicidal thoughts....but thats ok cause caster is suuuuuch a disgrace she deserves to have injections of synthetic hormones to CHANGE her bodys natural chemistry
but lets praise and applaud bruce jenner for taking PEDs to the point where he has a PSYCHOLOGICAL disorder and feels like he needs to dress and act like a woman.....liberals are so fukcing insane! caster has a GENETIC DISORDER and you crucify her, bruce jenner has a self induced PSYCHOLOGICAL DISORDER and you call him fukcing courageous?!?!
again READ and EDUCATE yourself, she is not a man pretending to be a woman, she is a woman with a GENETIC DISORDER, and 99% of these disorders there is adverse health effects and other problems, so in that case caster is a GENETIC freak in the fact she is not weak, febal, mentally challenged etc.....and since you liberal trolls cant click on links i will do the work for you (these are the intersex types and the issues)
1). Klinefelter syndrome (KS) also known as 47,XXY or XXY, is the set of symptoms that result from two or more X chromosomes in males.[1] The primary feature is sterility.[1] Often symptoms may be subtle and many people do not realize they are affected. Sometimes symptoms are more prominent and may include weaker muscles, greater height, poor coordination, less body hair, smaller genitals, breast growth, and less interest in sex.[2] Often it is only at puberty that these symptoms are noticed.[3] Intelligence is usually normal; however, reading difficulties and problems with speech are more common. Symptoms are typically more severe if three or more X chromosomes are present.[2]
Klinefelter syndrome usually occurs randomly. An older mother might increase the risk slightly. The condition is not inherited from one's parents.[4] The underlying mechanisms involves at least one extra X chromosome in addition to a Y chromosome such that there is a total of 47 or more chromosomes rather than usual 46.[5] KS is diagnosed by the genetic test known as a karyotype.[3]
2).Turner syndrome (TS) also known as Ullrich–Turner syndrome, gonadal dysgenesis, and 45,X, is a condition in which a female is partly or completely missing an X chromosome.[1] Signs and symptoms vary among those affected. Often, a short and webbed neck, low-set ears, low hairline at the back of the neck, short stature, and swollen hands and feet are seen at birth. Typically, they are without menstrual periods, do not develop breasts, and are unable to have children. Heart defects, diabetes, and low thyroid hormone occur more frequently. Most people with TS have normal intelligence. Many, however, have troubles with spatial visualization such as that needed for mathematics.[2] Vision and hearing problems occur more often.[3]
3). 45,X/46,XY mosaicism, also known as X0/XY mosaicism and mixed gonadal dysgenesis,[1] is a rare disorder of sex development in humans associated with sex chromosome aneuploidy and mosaicism of the Y chromosome. This is called a mosaic karyotype because, like tiles in mosaic floors or walls, there is more than one type of cell.
The clinical manifestations are highly variable, ranging from partial virilisation and ambiguous genitalia at birth, to patients with a completely male or female gonads. Most individuals with this karyotype have apparently normal male genitalia, and a minority with female genitalia, with a significant number of individuals showing genital abnormalities or intersex characteristics.[2] A significantly higher than normal number of other developmental abnormalities are also found in individuals with X0/XY mosaicism.[2] Psychomotor development is normal.
4). Smith–Lemli–Opitz syndrome (also SLOS, or 7-dehydrocholesterol reductase deficiency) is an inborn error of cholesterol synthesis.[1] It is an autosomal recessive, multiple malformation syndrome caused by a mutation in the enzyme 7-Dehydrocholesterol reductase, or DHCR7. It causes a broad spectrum of effects, ranging from mild intellectual disability and behavioural problems to lethal malformations.[2
Physical characteristics[edit]
The most common facial features of SLOS include microcephaly, bitemporal narrowing (reduced distance between temples), ptosis, a short and upturned nose, micrognathia, epicanthal folds, and capillary hemangioma of the nose.[3] Other physical characteristics include:
low-set and posteriorly rotated ears
high-arched, narrow, hard palate
cleft lip/palate
agenesis or hypoplasia of the corpus callosum
cerebellar hypoplasia
increased ventricular size
decreased frontal lobe size
polydactyly of hands or feet
short, proximally placed thumb
other finger malformations
syndactyly of second and third toes
ambiguous or female-like male genitalia
congenital heart defects
renal, pulmonary, liver and eye abnormalities
5). Lipoid congenital adrenal hyperplasia is an endocrine disorder that is an uncommon and potentially lethal form of congenital adrenal hyperplasia (CAH). It arises from defects in the earliest stages of steroid hormone synthesis: the transport of cholesterol into the mitochondria and the conversion of cholesterol to pregnenolone—the first step in the synthesis of all steroid hormones. Lipoid CAH causes mineralocorticoid deficiency in affected infants and children. Male infants are severely undervirilized causing their external genitalia to look feminine. The adrenals are large and filled with lipid globules derived from cholesterol.
Female patients[edit]
XX females with lipoid CAH may need estrogen replacement at or after puberty. Active intervention has been used to preserve the possibility of fertility and conception in lipoid CAH females.[6] In a case report in 2009, a woman with late onset lipoid CAH due to StAR deficiency underwent hormone replacement therapy in combination with an assisted fertility technique, intracytoplasmic sperm injection.[7] This led to ovulation and with implantation of the in vitro fertilized egg, a successful birth.
Male patients[edit]
Most XY children are so undervirilized that they are raised as girls. The testes are uniformly nonfunctional and undescended; they are removed when the diagnosis is made due to the risk of cancer development in these tissues.[8]
6). 3β-Hydroxysteroid dehydrogenase II deficient congenital adrenal hyperplasia (3β-HSD CAH) is an uncommon form of congenital adrenal hyperplasia (CAH) resulting from a mutation in the gene for one of the key enzymes in cortisol synthesis by the adrenal gland, 3β-hydroxysteroid dehydrogenase (3β-HSD) type II (HSD3B2).[1][2] As a result, higher levels of 17OH-pregnenolone appear in the blood with adrenocorticotropic hormone (ACTH) challenge, which stimulates adrenal corticosteroid synthesis.
There is a wide spectrum of clinical presentations of 3β-HSD CAH, from mild to severe forms. The uncommon severe form results from a complete loss of enzymatic activity and manifests itself in infancy as salt wasting due to the loss of mineralocorticoids. Milder forms resulting from incomplete loss of 3β-HSD type II function do not present with adrenal crisis, but can still produce virilization of genetically female infants and undervirilization of genetically male infants. As a result, this form of primary hypoadrenalism is the only form of CAH that can cause ambiguous genitalia in both genetic sexes.
7). Congenital adrenal hyperplasia due to 17α-hydroxylase deficiency is an uncommon form of congenital adrenal hyperplasia resulting from a defect in the gene CYP17A1, which encodes for the enzyme 17α-hydroxylase. It produces decreased synthesis of both cortisol and sex steroids, with resulting increase in mineralocorticoid production. Thus, common symptoms include mild hypocortisolism, ambiguous genitalia in genetic males or failure of the ovaries to function at puberty in genetic females, and hypokalemic hypertension (respectively).[1] However, partial (incomplete) deficiency is notable for having inconsistent symptoms between patients,[2] and affected genetic (XX) females may be wholly asymptomatic except for infertility.[3]
8). 17β-Hydroxysteroid dehydrogenase III deficiency is a rare disorder of sexual development affecting testosterone biosynthesis by 17β-hydroxysteroid dehydrogenase III (17β-HSD III), which can produce impaired virilization (traditionally termed male pseudohermaphroditism) of genetically male infants and children and excessive virilization of female adults.
It is an autosomal recessive[1] condition and is one of the few disorders of sexual development that can affect the primary and/or secondary sex characteristics of both males and females.
9). Swyer syndrome, or XY gonadal dysgenesis, is a type of hypogonadism in a person whose karyotype is 46,XY. The person is externally female with streak gonads, and if left untreated, will not experience puberty. Such gonads are typically surgically removed (as they have a significant risk of developing tumors) and a typical medical treatment would include hormone replacement therapy with female hormones.[1] The syndrome was named by Gerald Swyer, an endocrinologist, based in London, UK.
10). XX gonadal dysgenesis is a type of female hypogonadism in which no functional ovaries are present to induce puberty in an otherwise normal girl whose karyotype is found to be 46,XX. With nonfunctional streak ovaries she is low in estrogen levels (hypoestrogenic) and has high levels of FSH and LH. Estrogen and progesterone therapy is usually then commenced.
11). True hermaphroditism, also known as ovotesticular disorder of sex development, is a medical term for an intersex condition in which an individual is born with ovarian and testicular tissue. There may be an ovary underneath one testicle or the other, but more commonly one or both gonads is an ovotestis containing both types of tissue.
Although similar in some ways to mixed gonadal dysgenesis, the conditions can be distinguished histologically.[1]
there are a number of other rare syndromes, and as you can see all of them are genetic disorders, not some psychological disorder of some libarl dude head case "identifying" as a woman. more than likely caster will need to surgically remove the undeveloped internal testes cause it says those usually turn cancerous. how is this so HARD for you idiots to understand? oh i know how cause liberals BELIEVE their own opinions as if it was the word of God instead of the truth and reading and educating themselves.
i think shannon and the rest of the girls need to educate themselves before they open their mouth in ignorance and stupidity. this isnt bruce jenner in his prime dressing up like a girl to win the gold, no its a human being that was born with a GENETIC DISORDER, she has a vagina from birth....how is that cheating? put her in the "special" olympics? unbelievable