Oh c'mon.
In this thread - and in discussions about this topic generally - the issue is not about female athletes who are doping on exogenous T. The issue is the natural levels of endogenous T of athletes competing in women's athletics.
Specifically, it's about the natural levels of endogenous T of XY DSD athletes with male androgen receptors whose natural T is in the male range because it comes from their testes versus the natural levels of endogenous T of XX female athletes with female androgen receptors whose natural T is in the female range because it comes from their ovaries, adrenal glands and fat.
I said: "In males, natural T made by the testes is the major controller and driver of development and a key factor in health, fitness and wellbeing at crucial stages of life - during gestation in utero, the male mini puberty of infancy in the first year of life, and puberty of adolescence and beyond. But natural T made by females - only half of which comes from the ovaries, the other half of female T comes from the adrenals and fat - appears to play relatively little role in female development, physiology, anatomy and sports performance."
In response, you linked to an article about a study in which "48 healthy 18- to 35-year-old women were randomly assigned to 10 weeks of daily treatment with10mg of testosterone cream or 10mg of a placebo."
Then you say "it is findings like this which lead me to wonder if unusually high levels of endogenous testosterone are more beneficial, and common among unambiguously female athletes, than we realize, and that the androgen gap to the DSD athletes is not quite as wide as we imagine."
Bringing the issue of women taking exogenous T into the convo is clearly just an attempt to muddy the waters and sow confusion.
There is no question that female athletes who take exogenous T obtain physical benefits from it that improve sports performance at least in the short and medium term. That's been proven incontrovertibly time and again by the example of numerous individual athletes over the course of decades - and by the large-scale doping of elite female athletes by former Soviet bloc countries like the former GDR in the 1960s, 70s and 80s.
It's also been shown more recently by research on females who take exogenous T to try to match male levels for purposes of "gender transition."
But women who have levels of T above the normal female range because they are taking exogenous T manufactured by the pharmaceutical industry are not at all analogous with, or comparable to, women who have levels of T above the normal female range because they have something going in their bodies that's causing an over-production of natural, endogenous T by their ovaries, adrenals and/or fat cells.
For the umpteenth time: when female persons have levels of natural T elevated beyond the normal female range (0.02 - 1.7 nmol/L), it is always because of a complex medical disorder or disease - PCOS, CAH, LOCAH, Cushing's disease, cancer - or because of pregnancy, which is a complex medical condition but not a disorder or disease.
All of these conditions have wide-ranging impacts on myriad organs, body systems and overall health. None of these medical conditions leads to or is associated with the kinds of tip-tip shape and peak physical fitness required for elite sports performance; and none are known to result in improved performance in elite track and field. Not even the healthiest, most physically-fit women going through the healthiest pregnancies imaginable do better in track and other sports because of the elevated levels of natural T that come with pregnancy.
Moreover, any female person who has natural, endogenous T even approaching the bottom end of the range that's customary for the XY DSD athletes competing in women's elite athletics would need urgent evaluation for a life-threatening endocrine tumor. But even a woman with a testosterone-secreting tumor or bunch of tumors would not have natural T anywhere near the level of most XY DSD athletes in women's sports because they usually have natural T levels in the middle to high end of the normal male range. In fact, adults with Caster Semenya's DSD, XY 5-ARD, often have natural T considerably exceeding the normal male range.