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RE: LYDIARD OR DANIELS?
The quoted information in the previous post was from Dr. Jens Bangsbo and Carsten Juel of the Institute of Exercise and Sports Sciences at the Cpenhage Muscle Research Centre (Institute of Molecular Biology and Physiology), University of Copenhagen, Denmark who were proving counter-point information to Drs.Lamb and Stephenson.
I will say that after reading their arguments, Dr. Lamb and Stephenson (their foe) seem to be making assumptions based on isolation-methods that are not uniform with real intra-cellular circumstances in working muscle.(Note I am not a biochemist and this is my layman's interpretation.)
One interesting point that Drs Bangbo and Juel make refutes the idea that lactate is a "savior" that reduces fatigue:
"...a number of studies have shown that lowered pH leads to greater potassium release and potassium accumulatin in the muscle interstitium..."
My analysis: The above statment leads to the obvious conclusion that electrolyte imbalance occurs as lactate rises; thus, a reduction of muscle contractile force and velocity of contraction occurs. When potassium is released in excess and accumulatess in the muscle interstitium, the sodium-potassium pumps will be directly influenced: contraction is limited, reduced, or concluded when lactate rises too high.
Pragmatics: If an athlete runs reps fast and in high volume with insufficient recovery a point is reached where it is impossible to keep the speed going.
That's obvious enough to any runner who has done fast rep workouts, but to the pseudo-scientists who insists that lactate is a "friend" is in denial of the reality. A coach like Lydiard, trained by the German scientists, who were doing the original lactate testing, knew that accumulating high lactate levels reduced capacity to run fast in sustained fashion, and training to elevate the "cruising speed" above which accumulating lacate occurs is a vital element of performacne succes. *And, once cruising speed is raised through voluminous distance running, it is possible to do large volumes of high-speed repetition training; hence providing increased capacity to deal with lactate accumulation and associated acidosis ONCE it occurs.
From Bangsbo and Juel, as they argue against making conclusions based on unrealistic situations:
"The argument for a positive role of lactic acid are based on studies of isolated and noncontracting muscle and skinned muscle fibers. In our review we argued that these results represented ARTIFICIAL nonexercise-related conditions and it is NOT possible to extrapolate to the in vivo condition (reality).
Further, they conclude:
"The capacity for lactate/H+ transport in human muscle is incrased by training and there is a positive correlation between transport capacity (i.e. efflux) and performance (Piligaard H, 1994); these findings further suggest that lactic acid accumulation is a DISADVANTAGE."
Yeah, I know you may have a continued problem with the word lactic acid, but the physicochemical argument defines the process as occuring (but rapid) - in the water milieu of the cells being identified. Just because it is instantaneous doesn't mean that it doesn't result nor does it take away the fact that acidosis occurs.
No matter how you slice it, lactate accumulation is NOT disinvolved with fatigue!
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