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RE: LYDIARD OR DANIELS?
The quoted information in the previous post was from Dr. Jens Bangsbo and Carsten Jeul of the Institute of Exercise and Sports Sciences at the Cpenhage Muscle Research Centre (Institute of Molecular Biology and Physiology), University of Copenhagen, Denmark who were proving counter-point information to Drs.Lamb and Stephenson.
I will say that after reading their arguments, Dr. Lamb and Stephenson seem to be making assumptions based on isolation methods that are not uniform with real intra-cellular activities. Note I am not a biochemist and this is my face-value observation.
One interesting point that Drs Bangbo and Juel make refutes the idea, at the least indirectly, that lactate is the "savior" that reduces fatigue:
"...a number of studies have shown that lowered pH leads to greater potassium release and potassium accumulatin in the muscle interstitium..."
The above statment leads to the fairly obvious conclusion that electrolyte imbalance occurs as lactate rises and, thus, a reduction of muscle contractile force and velocity of contraction. When potassium is released in excess and accumulatess in the muscle interstitium, the sodium-potassium pumps will be directly influenced - inhibited to work well. That simple means contraction is limited, reduced, or concluded when lactate rises to high.
So,if you are a runner and you run reps fast and in high volume with insufficient recovery you'll get to a point where simply can't keep the speed going. That's obvious enough to any runner who has done fast rep workouts, but to the pseudo-scientists who insist that lactate is a "friend" is in denial of the reality. A coach like Lydiard, trained by the German scientists who were doing the lactate testing, knew that a accumulating a high level of lactate was reducing capacity to run fast and that training to elevate the cruising speed where running doesn't accumulate lacate is vital to succes. And, once cruising speed is raised, doing a high volume of high-speed repetition trainng provides increased capacity to deal with lactate accumulation ONCE it occurs.
From Bangsbo and Jeul as they argue against making conclusions, as many have, that are based on unrealistic situations:
"The argument for a positive role of lactic acid are based on studies of isolated and noncontracting muscle and skinned muscle fibers. In our review we argued that thes results represented ARTIFICIAL nonexercise-related conditions and it is NOT possible to extrapolate to the in vivo condition (reality).
Further, they conclude:
"The capacit for lactate/H+ transport in human muscle is incrased by training and there is a positive correlation between transport capacity (i.e. efflux) and performance (Piligaard H, 1994); these findings further suggest that lactic acid accumulatin is a DISADVANTAGE."
Yeah, I know you may have a continued problem with the word lactic acid, but when you look at the physicochemical argument you realized that it is indeed present but does change very rapidly in the water milieu of the cells being identified. That doesn't mean that it doesn't happen though. And, no matter how you slice it, lactate accumulation is not disinvolved with fatigue!
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