trollism wrote:
Can't get sued for expressing an opinion, particularly where the opinion is a fair one despite it being controversial.
Saying that you think an distance running record (a sport riddled with PEDs) is a bit sketchy is a perfectly valid opinion to have.
Saying he saw an athlete inject EPO into her ass (when he didn't or has no proof that he did) is libel. Saying that XXX athlete might be doped, is not.
trollism wrote:Can I do it unkindly instead?
I want to use the words 'moron', 'idiot' and 'imbecile' as liberally as you like to.
ventolin^3 wrote:
trollism wrote:Can I do it unkindly instead?
I want to use the words 'moron', 'idiot' and 'imbecile' as liberally as you like to.
moron
the question stands :
kindly inform us of an
"unproven"
doping country ???
To the Limit wrote:Ha ha!
The Uk has a far better record in terms of lack of drug users in middle and distance running than Ethiopia, as well as having had far more stringent and vigorous testing since the early 80's.
ventolin^3 wrote:
moron
rojo wrote:One thing we hinted at but didn't say directly is based on the equivalent performance charts - women are way better at longer distance stuff than shorter distance stuff. I dont know if the charts are made for men or what but they don't really work great for women - the longer they the, the better their records get
just sayin wrote:
To the Limit wrote:
Ha ha!
The Uk has a far better record in terms of lack of drug users in middle and distance running than Ethiopia, as well as having had far more stringent and vigorous testing since the early 80's.
the british are very crafty when it comes to doping
they don't get caught much
ventolin^3 wrote:
the chances coe caught toxoplasmosis from scooping cat sh!t/dodgy takeaway/contaminated meat or water are virtually nil
ventolin^3 wrote:
eh ?
the chances coe caught toxoplasmosis from scooping cat sh!t/dodgy takeaway/contaminated meat or water are virtually nil
try this :
http://www.nlm.nih.gov/medlineplus/toxoplasmosis.html
An actual doctor wrote:
ventolin^3 wrote:
eh ?
the chances coe caught toxoplasmosis from scooping cat sh!t/dodgy takeaway/contaminated meat or water are virtually nil
try this :
http://www.nlm.nih.gov/medlineplus/toxoplasmosis.html
You and your various other handles are collectively dumb MFs. Call yourself a doctor. Sh!thead.
T gondii oocysts, tachyzoites, and bradyzoites can cause infection in humans. Infection can occur by ingestion of oocysts following the handling of contaminated soil or cat litter or through the consumption of contaminated water or food sources (eg, unwashed garden vegetables). Transmission of tachyzoites to the fetus can occur via the placenta following primary maternal infection.
RARELY, infection by tachyzoites occurs from ingestion of unpasteurized milk or by direct entry into the bloodstream through a blood transfusion or laboratory accident. Transmission can also occur via ingestion of tissue cysts (bradyzoites) in undercooked or uncooked meat or through transplantation of an organ that contains tissue cysts. (Slaughterhouse workers and butchers may be at increased risk of infection.) In Europe and the United States, pork is the major source of T gondii infection in humans.
The seroprevalence of T gondii antibodies in the human population varies geographically, with prevalence rates approaching 90% in some European countries.
Not a Coach wrote:
ventolin^3 wrote:
the chances coe caught toxoplasmosis from scooping cat sh!t/dodgy takeaway/contaminated meat or water are virtually nil
Which may explain why it took so long to diagnose
An actual doctor wrote:You and your various other handles are collectively dumb MFs. Call yourself a doctor. Sh!thead.
T gondii oocysts, tachyzoites, and bradyzoites can cause infection in humans. Infection can occur by ingestion of oocysts following the handling of contaminated soil or cat litter or through the consumption of contaminated water or food sources (eg, unwashed garden vegetables). Transmission of tachyzoites to the fetus can occur via the placenta following primary maternal infection.
RARELY, infection by tachyzoites occurs from ingestion of unpasteurized milk or by direct entry into the bloodstream through a blood transfusion or laboratory accident. Transmission can also occur via ingestion of tissue cysts (bradyzoites) in undercooked or uncooked meat or through transplantation of an organ that contains tissue cysts. (Slaughterhouse workers and butchers may be at increased risk of infection.) In Europe and the United States, pork is the major source of T gondii infection in humans.
The seroprevalence of T gondii antibodies in the human population varies geographically, with prevalence rates approaching 90% in some European countries.
An actual doctor wrote:Dear Venty,
The person who is "not too bright" would be you. I explained above the myriad ways you could get toxo in an area, Europe, where large numbers of the population have antibodies to toxo, indicating past exposure. The vast majority do not develop clinical disease
Coe travelled and raced extensively in Europe (especially as the UK happens to be part of, you guessed it, Europe). If his immune system was banged about by the extremely hard training he did, he could have become ill with toxo, instead of just being an asymptomatic carrier
Or perhaps you would prefer to believe he has AIDS to account for an immune deficiency
An actual doctor wrote:
Dear Venty,
Yes, I posted from MedScape, an extremely reputable website written by medical specialists for doctors generally. I actually understood what I read, though. So I figured out that the chance of Coe being exposed to toxo through the environment was sufficiently high to make the assertion he got it from a blood transfusion to fanciful.
