wellnow wrote:
No, you're still missing the point. You and the PhD are trying to prove a hypothesis about increased RBCs being the ideal and the performance improver.
If Iron status is where it should be, RBC mass will be where it should be. Improvement come from better speed endurance. The blood boost hypothesis is pure dogma.
Well dogma or not, it's the mainstream opinion in ex phys. If gains in RBCs don't affect performance, why does EPO work so well? That's basically what altitude does (as documented by Stray-Gundersen)--it causes a spike in blood EPO levels, which stimulates RBC production and enhances performance via increased oxygen-carrying capabilities.
I have two questions for you, then:
1) If RBC increases don't account for improved performance, why does EPO work?
2) What is the physiological adaption from live-high-train-low training that DOES increase race performance? It is fairly clear that live-high-train-low WORKS. So if not RBCs, then what?